The ability of some HIV 1-infected individuals to maintain plasma virus levels of < 50 copies/ml and normal CD4 counts without therapy appears to be related to the indivi
The battle between the human immunodeficiency virus (HIV) and the host is a complex game of chess in which one tries to evade and conquer the other.
While the HIV virus evolves to find new ways to avoid detection and destruction by the host's immune system, host gene mutations have been identified in some individuals that prevent CD4 receptor attachment so that the virus cannot gain entry.
Recently, genetic make-up was found to influence protection against the human immunodeficiency virus (HIV) and other foreign invaders. Individuals who carry the gene for expression of HLA-B57 antigens have been shown to have much slower HIV-1 (human immunodeficiency virus type 1) disease progression than individuals who do not express HLA-B57.
Read more about HIV infection and protection:
HIV Protection: CCR5 Chemokine Receptor Mutation
Resistance to Anti-viral Agents: Mutations Side-step Inhibition Processes
